
参考资料
[1] 中华人民共和国国家卫生健康委员会, 中华人民共和国卫生行业标准-高尿酸血症与痛风患者膳食指导(WS/T 560-2017), 中华人民共和国国家卫生健康委员会
[2] Mark A. Herman and Morris J. Birnbaum. Molecular Aspects of Fructose Metabolism and Metabolic Disease. Cell Metab. 2021 Dec 7; 33(12): 2329–2354.
[3] Congwang Zhang a, Lijun Li b, Recent advances in fructose intake and risk of hyperuricemia. Biomedicine & Pharmacotherapy. Volume 131, November 2020, 110795.
[4] Takahiko Nakagawa, Miguel A Lanaspa, Richard J Johnson. The effects of fruit consumption in patients with hyperuricaemia or gout. Rheumatology, Volume 58, Issue 7, July 2019, Pages 1133–1141.
[5] Mattia Coronati, Francesco Baratta, et al. Added Fructose in Non-Alcoholic Fatty Liver Disease and in Metabolic Syndrome: A Narrative Review. Nutrients. 2022 Mar 8;14(6):1127. doi: 10.3390/nu14061127.
[6] Johnson R J, Stenvinkel P, Andrews P, et al. Fructose metabolism as a common evolutionary pathway of survival associated with climate change, food shortage and droughts[J]. Journal of Internal Medicine, 2019. doi/10.1111/joim.12993
[7] Joseph Jamnik, Sara Rehman, et al., Fructose intake and risk of gout and hyperuricemia: a systematic review and meta-analysis of prospective cohort studies. Nutrition and metabolism
[8] Soraiya Ebrahimpour-koujan, Parvane Saneei, et al., Consumption of sugar sweetened beverages and dietary fructose in relation to risk of gout and hyperuricemia: a systematic review and metaanalysis. Critical Reviews in Food Science and Nutrition, DOI: 10.1080/10408398.2018.1503155
